A novel Bcl-2 inhibitor, BM-1197, induces apoptosis in malignant lymphoma cells through the endogenous apoptotic pathway | BMC Cancer | Full Text
A) Bcl-2 family member effector proteins (Bax and Bak) oligomerize and... | Download Scientific Diagram
Cells | Free Full-Text | Restoring Apoptosis with BH3 Mimetics in Mature B-Cell Malignancies
BCL‐2‐family protein tBID can act as a BAX‐like effector of apoptosis | The EMBO Journal
A model for PUMA-mediated apoptosis. PUMA activates Bax/Bak indirectly... | Download Scientific Diagram
Bcl-2 - Wikipedia
Interplay of Bcl-2 Proteins Decides the Life or Death Fate ~ Fulltext
Mechanism of apoptosis induction by inhibition of the anti-apoptotic BCL-2 proteins | PNAS
PUMA-mediated apoptosis. PUMA is induced by a wide range of death... | Download Scientific Diagram
BH3-only sensors Bad, Noxa and Puma are Key Regulators of Tacaribe virus-induced Apoptosis | PLOS Pathogens
Efficacy and specificity of inhibitors of BCL-2 family protein interactions assessed by affinity measurements in live cells | Science Advances
The BCL-2 protein family, BH3-mimetics and cancer therapy | Cell Death & Differentiation
Ancient and conserved functional interplay between Bcl-2 family proteins in the mitochondrial pathway of apoptosis | Science Advances
Targeting BCL-2 regulated apoptosis in cancer | Open Biology
Basal Patologi KU - BAD, BID and PUMA proteins shift the balance between the pro-apoptotic and anti-apoptotic family members by neutralizing the actions of anti-apoptotic proteins like BCL2 and BCL-XL, thereby promoting
Co-Operativity between MYC and BCL-2 Pro-Survival Proteins in Cancer. - Abstract - Europe PMC
Cooperation of BH3-only proteins in killing neurons
Why do BCL-2 inhibitors work and where should we use them in the clinic? | Cell Death & Differentiation
Targeting BCL-2 in B-cell malignancies and overcoming therapeutic resistance | Cell Death & Disease
Targeting BCL-2 regulated apoptosis in cancer | Open Biology
Dysregulation of BCL-2 family proteins by leukemia fusion genes
PUMA decreases the growth of prostate cancer PC-3 cells independent of p53